Rickets is a deficiency in calcium and phosphate ions which mainly affects children. May this be mainly due to a lack of vitamin D or a lack of calcium and phosphate in the diet. Lack of exposure to sunlight May also cause rickets, because the active form of vitamin D, which is vitamin D3, requires activation by ultraviolet light. Without it, remains largely unnecessary vitamin D and calcium and phosphate absorption is still insufficient.

Children who stay at home during the winter in May suffers from rickets in the spring. Because the liver stores enough vitamin D during the summer months and are trained enough to take the child through the winter. In spring, the stores of vitamin D in May and short May develop rickets.

Parathyroid hormone in rickets

Due to the decrease in plasma concentration of calcium in rickets, has stimulated the parathyroid glands are parathyroid hormone secretion rate of growth. While this May help to increase the plasma concentrations of calcium, phosphate in the plasma concentrations in May continue to fall rapidly.

This is due to the action of parathyroid hormone on the kidneys. Although parathyroid hormone prevents the excretion of calcium ions by the kidneys, there is a loss of urine by the phosphate ions yet. For this reason, plasma concentrations of calcium only slightly below normal for early May rickets, while there is a lack of phosphate ions.

In rickets extended, however, a weakness in bones and bone deformities occur in May. Compensatory growth of the parathyroid hormone in response to low calcium ion and phosphate at the end of the valve remains in addition to the supply. Bone to grow weaker and weaker as the parathyroid hormone valves in the rich source of calcium to meet the needs of the organization. While osteoblasts can continue to make new layers of bone growth in children, new bone is largely uncalcified is still low and normal bone.

Finally, even the bones would not be able to support the body of calcium and without treatment, tetany may occur. Tetany May be imposed at the beginning of rickets because osteoclasts can still exploit the bones of the bones in the body needs calcium. In prolonged rickets, the bones were exhausted in May of calcium and the body can not get the calcium needed. When this happens, a child with rickets May tetanus die from respiratory spasms if not administered by intravenous calcium.

Treatment of rickets

The treatment of rickets based mainly on a diet to meet calcium and phosphate. Equally important is the addition of vitamin D in food to help absorption of calcium and phosphate in the intestine. So little vitamin D is administered, there is minimal absorption of necessary nutrients and rickets can still occur.

For skeletal deformities, positioning and timing can help. May severe deformities require surgical correction. The best treatment for rickets, however, is still prevention. Make sure you and your children get enough sun and enough calcium and vitamin D in your diet.